SOCIAL IDENTITY, SOCIAL CHANGE AND THE CONSTRUCTION OF SYMBOLIC BOUNDARIES IN A WEST HIGHLAND SETTLEMENT

Merged with duplicate record 10026.1/1620 on 28.02.2017 by CS (TIS)In the last thirty years there have been very few systematic observations and analyses of everyday life at a local level. (see Bell and Newby, 1972) As a result our knowledge of local sub-cultures is seriously outdated (E. S. R. C., 1982,13) and we are not maintaining an up to date social history of our own times. However, Bulmer, in an article entitled "The Rejuvenation of Community Studies? " (1985) and Willmott (1986) have both identified "some sign that the study of localities is being revived". (Bulmer, 1985,433) This thesis, a sociological account of contemporary rural life in part of the Scottish Highlands, is a distinctive contribution to that revival. The analysis proceeds on a number of levels and shows several signs of originality. It is more than a simple ethnography. By examining change and social process it goes beyond previous static and structural studies to analyse the implications that indigenous perceptions of identity and 'belonging' have for the nature of social process in a particular locality. Important differences from other localities and locality studies are recognised and social identity, rather than social structure, is the key element in explaining people's involvement in social change and the processes of daily life, and is the central issue around which the thesis develops. Analysing the division that exists between 'locals' and 'incomers' in Fearnbeg involves operating at an advanced level with the interaction of symbols and actions. (While this is not a new challenge, its manifestation with regard to this location is unique). The thesis demonstrates that the most important social division in Fearnbeg society cannot be explained in structural terms alone. The core dicthotomy, and why Fearnbeg people think and behave in the ways they do, can only be understood in terms of cultural and symbolic boundaries

The life and teaching of Robert Hall, 1764-1831

No abstract supplied

Co-morbidity burden in Parkinson’s disease : Comparison with controls and its influence on prognosis

Financial support This study was funded by Parkinson’s UK, the Scottish Chief Scientist Office, NHS Grampian endowments, the BMA Doris Hillier award, RS Macdonald Trust, the BUPA Foundation, and SPRING. The funders had no involvement in the study. Acknowledgements We acknowledge funding for the PINE study from Parkinson’s UK (G-0502, G-0914, G-1302), the Scottish Chief Scientist Office(CAF/12/05), the BMA Doris Hillier award, RS Macdonald Trust, the BUPA Foundation, NHS Grampian endowments and SPRING. We thank the patients and controls for their participation and the research staff who collected data and supported the study database.Peer reviewedPostprintPostprintPublisher PD

End of British rule in South Arabia, 1959-1967

This thesis analyses British policy in the final years of colonial rule in Aden and the Aden Protectorate (South Arabia), the period 1959 to 1967. This work deals first with the first century of British rule in Aden, from the capture of the port in 1839 until the end of the Second World War in 1945, examining the role of the Colony in British overseas policy. Secondly, the thesis gives the international and regional background to the period in question by giving a summary of British overseas policy, the Cold War and the Arab Cold War in the period 1945 to 1967. Thirdly, it tackles the history of colonial rule between 1945 and 1959, covering the increasing value of Aden to British defence policy in the Middle East, as well as the creation of a Federation among the local rulers in an attempt to bolster Britain's closest allies in South Arabia. The fourth point of the thesis is the examination of British defence policy, 1959 to 1963, which saw the military base in Aden become vital to London's overseas policy. This period saw Aden merge with the Federation, against a background of opposition from Arab Nationalists, in an attempt to secure British interests. Fifthly, the thesis analyses the gradual loss of British control over events in Aden and the Federation as the Arab Nationalist campaign became increasingly effective. The British Government finally decided to grant independence to appease the opposition, but retain the base for the defence of Britain's overseas interests. The thesis then attempts to chart the rise of the eventual victors in the conflict, the 'Marxist' National Liberation Front and its rivalry with other Arab Nationalist groups. Finally, the thesis examines the final period of British rule in Aden, from the Defence White Paper of February 1966, when the decision was taken to cut many of Britain's overseas commitments, including the base in Aden, to the withdrawal of November 1967. This period saw the disintegration of the Federation and the inability of the British to prevent the Nationalists taking power. The thesis concludes that towards the end of colonial rule, British policy in South Arabia was incoherent and suffered from division among the different Government departments. Furthermore, the inability to protect the Federation effectively enabled the Nationalists to undermine Britain's only allies in the Protectorate

The genetics of drug resistance in malaria parasites

This thesis describes work performed on the genetics of resistance to the drugs sulphadiazine and pyrimethamine in the rodent 'malaria parasite Plasmodium chabaudi.A number of lines resistant to pyrimethamine were obtained by direct exposure to the drug in a single step. Characteristically these were some 20 times resistant to pyrimethamine and had an increased dependence on FABA. Subsequent genetic analyses showed that this class of mutant was genetically homogeneous and that the primary resistance to pyrimethamine and the concomitant FABA dependence were inseparable by recombination — both consistent with a single gene model of resistance.a number of lines resistant to sulphadiazine were obtained in a single step either by direct exposure to the drug or following FABA restriction. Characteristically these lines were some 4 times resistant to sulphddiazine and required a reduced FABA growth supplement but exhibited a variable spectrum of crossresistance to pyrimethamine uncorrelated with their mode of selection. A phenotypically indistinguishable example of this type of line was also obtained following exposure to pyrimethamine. Evidence from subsequent selection experiments was taken to show that at least two types of separate heritable change might result in the acquisition of sulphadiazine 1' G kj 1 £> ticlaiC G 0 ai-Xl GUxcLmple of one of these was later shown to segregate in a simple mendelian fashion when crossed with a wild line.A number of lines were obtained by selection in two discrete steps: either following the FABA restriction of a pyrimethamine resistant line; or by the direct exposure of a sulphadiazine resistant line to pyrimethamine. While these lines uere some 40 times resistant to pyrimethamine they exhibited wild type responses to. sulphuuiazine and PABA.One such line was crossed with a wild line and two types of non-parental clone were detected among the-products of the cross. These resembled either the pyrimethamine resistant or sulphadiazine resistant lines obtained by selection in a single step. This result was consistent with a genetic recombination event having taken place between, the genetic elements proposed to confer resistance to either of these two drugs. Evidence from a reconstruction experiment suggested that the linkage between these elements was either loose or non¬ existent. \An unsuccessful attempt was mq.de to analyse this cross in the greater precision afforded by the characterisation of clones derived from single oocysts obtained by micro¬ dissection. ) In another cross between two single step lines resistant to pyrimethamine and to sulphadiazine respectively both wild type and double mutant parasites were detected among the cross—products - thus confirming that recombination could link these mutants to produce multiple drug resistance.during the development of a rat-adapted line of P.chabaudi (which was later shown to be unsuitable for use as a selective marker) it was discovered that garnetocytogenesis was enhanced in unadapted parasites during subsistence in rats. This led to the substitution of rats for mice as rodent carriers of the foi-parental mixed-blood infection prior to cyclical passage in crosses.Estimates of the rates of acquisition are presented for each type of resistance in the Discussion together with a critic appreciation of the extent to which these represent true mutation rates

Associations between antiepileptic use and hypogammaglobulinemia : Findings from a population-based case-control study using data linkage.

Acknowledgments We acknowledge the support of the Grampian Data Safe Haven (DaSH) facility within the Aberdeen Centre for Health Data Science and the associated financial support of the University of Aberdeen and NHS Research Scotland. We are also grateful for funding from the School of Medicine, Medical Sciences, and Nutrition, University of Aberdeen. The funders had no role in the execution or design of this study. Funding statement: We are grateful for funding for data linkage from the School of Medicine, Medical Sciences, and Nutrition, University of Aberdeen. Funders had no role in the execution or design of this study.Peer reviewedPublisher PD

Associations between antiepileptic use and hypogammaglobulinemia : Findings from a population-based case-control study using data linkage.

Funding statement: We are grateful for funding for data linkage from the School of Medicine, Medical Sciences, and Nutrition, University of Aberdeen. Funders had no role in the execution or design of this study.Peer reviewedPostprin